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One study suggests that the mood and behavioral effects seen during anabolic-androgenic steroid misuse may result from secondary hormonal changesin the brain. It is possible that these changes in the brain may result from altered neurotransmission in the nucleus accumbens and amygdala, areas implicated in mood regulation. A study by Vrana et al. (1985) evaluated the antidepressant effect of a selective androgen receptor antagonist in female human subjects. In this study, female patients who had been treated with cyproterone acetate experienced a reduction in depression while on this treatment compared to similar subjects given placebo. The difference could be attributed to treatment with a selective atrogen receptor antagonist. A recent study of depressed male subjects has found that the administration of testosterone to rats reduces depression, whereas only a single injection of androstenedione reduces depression in men (Iyer et al., 2008). The above studies have indicated that androgenic steroid abuse can increase depression and anxiety. It is important that the general population take care of symptoms that result from steroids like decreased energy, mood alterations, increased cortisol and adrenaline levels, decreased appetite and excessive hunger, and more often than not, increased risk-taking behavior (Alford & Hallett, 2002). This is why anti-anxiety medication is necessary. 3. Antidepressants: In a study of female rats administered a high dose of androstenedione, there was increased anxiety and depression over a period of three days following an injection. When compared to the group which received vehicle, a significant increase of anxiety was observed in both the androgen-treated groups and there were significantly increased depression in the androgen-treated groups. A study has been conducted on male rats, where it has been found that testosterone has neuroprotective effects. This is an effect which is believed to occur due to androgen antagonism. This study utilized the administration of testosterone enanthate to male rats, as previously mentioned, with no androgen administration. It has been found that testosterone therapy is not effective when rats are under stress. In fact, it has been found that there is an increased risk of side effects associated with testosterone administration in certain stress-reactivity studies in the literature. In other words, if stress is present in the study then there is an increased risk of adverse effects. It does not make sense to treat a stressed animal with testosterone while there is a possibility of overstimulation in their nervous system. A similar study in male rats has been conducted in which the administration of testosterone was discontinued following an injury to the rats. This study showed that testosterone therapy was effective in Similar articles:
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